I'm not sure why I made a not of this one now, but I wondered if mcb1 is definitely
GO detection of DNA damage stimulus involved in mitotic G2 DNA damage checkpoint
(this would make it a 'sensor' for the checkpoint)
rather than just causing a problem which results in activation of the checkpoint.
I think we only know that arrest requires that the chk1 checkpoint is functional
Thoughts?
I haven't dealt directly with the paper cited for that annotation, but I don't know of any other reasons to prefer a GO annotation over just phenotypes for mcb1 and checkpoints.
Ah Ok, it was me!. I deleted the GO annotation but i 'll double check with Jacky.
I have a not on my list to discuss phenotype vs. GO annotation with here so this will be a good example.
Diff: